Which technologies and conditions made world war i a new kind of war? select all that apply.

1. On the history of the battle at Ypres and its relationship to military history and the history of science and medicine, see Smart Jeffery K., “History of Chemical and Biological Warfare: An American Perspective,” in Medical Aspects of Chemical and Biological Warfare, ed. Frederick R. Sidell, Ernest T. Takafuji, and David R. Franz (Washington, DC: Office of the Surgeon General, 1997), 15; L. F. Haber, The Poisonous Cloud: Chemical Warfare in the First World War (New York: Clarendon Press, 1986), 31–32. On the general topic of chemical weapons history in the United States, see Leo B. Brophy, Wyndham D. Miles, and Rexmond C. Cohrane, The Chemical Warfare Service: From Laboratory to Field (Washington, DC: Office of the Chief of Military History, Department of the Army, 1959); Brooks E. Kleber and Dale Birdsell, The Chemical Warfare Service: Chemicals in Combat (Washington, DC: Center for Military History, United States Army, 2003); Leo P. Brophy and George J.B. Fisher, The Chemical Warfare Service: Organizing for War (Washington, DC: Center of Military History, United States Army, 2004). On the cultural, political, and scientific history of chemical warfare in the Unites States, see Edmund P. Russell III, “ ‘Speaking of Annihilation’: Mobilizing for War Against Human and Insect Enemies, 1914–1945,” Journal of American History 82 (March 1996): 1505–1529; see also by Russell, War and Nature: Fighting Humans and Insects With Chemicals From World War I to Silent Spring (New York, NY: Cambridge University Press, 2001); Hugh Slotten, “Humane Chemistry or Scientific Barbarism? American Responses to World War I Poison Gas, 1915–1930,” Journal of American History 77 (September 1990): 476–498; William L. Sibert, “Chemical Warfare,” Journal of Industrial and Engineering Chemistry 11 (November 1919): 1060–1062; Newton D. Baker, “Chemistry in Warfare,” Journal of Industrial and Engineering Chemistry 11 (September 1919): 921–923.

2. Joy Robert J.T., “Historical Aspects of Medical Defense Against Chemical Warfare,” in Medical Aspects of Chemical and Biological Warfare, 90.

3. Watkins O. S., Methodist Report, cited in Amos Fries and C. J. West, Chemical Warfare (New York: McGraw Hill, 1921), 13; also cited in Joy, “Historical Aspects of Medical Defense,” 90.

4. For a very useful and up-to-date introduction to chemical weapons and chemical terrorism, see Tucker Jonathan B., “Introduction,” in Toxic Terror: Assessing Terror Use of Chemical and Biological Weapons, ed. Jonathan B. Tucker (Cambridge, MA: MIT Press, 2000), 3. Tucker further explains the various classifications of chemical agents, which include choking agents that damage lung tissue (e.g., chlorine, phosgene), blood agents that interfere with cellular respiration (e.g., hydrogen cyanide), blister agents that cause severe chemical burns to the skin and lungs (e.g., mustard gas, lewisite), and nerve agents that disrupt nerve-impulse transmission in the central and peripheral nervous systems, causing convulsions and death by respiratory paralysis (e.g., sarin, VX).” Tucker, 33.

5. For an introduction to war, medicine, and public health, see War and Public Health, ed. Barry S. Levy and Victor W. Sidel (Washington, DC: American Public Health Association, 2000), especially the following articles: William H. Foege, “Arms and Public Health: A Global Perspective” (3–11); Richard M. Garfield and Alfred I. Neugut, “The Human Consequences of War” (pp. 27–38). On the specific topic of chemical weapons, see Allan H. Lockwood, “The Public Health Effects of the Use of Chemical Weapons,” in War and Public Health, 84–97; Challenges in Military Health Care: Perspectives on Health Status and the Provision of Care, ed. Jay Stanley and John D. Blair (New Brunswick, NJ: Transaction Publishers, 1993). On current military–medical approaches to the threat of chemical warfare and viable medical responses to that threat, see Ernest R. Takfuji and Allart B. Kok, “The Chemical Warfare Threat and the Military Healthcare Provider,” in Medical Aspects of Chemical and Biological Warfare, 111–128; Ernest T. Takfuji, Anna Johnson-Winegar, and Russ Zajtchuk, “Medical Challenges in Chemical and Biological Defense for the 21st Century,” in Medical Aspects of Chemical and Biological Warfare, 667–686.

6. Historians continue to debate the exact number of casualties incurred during World War I, because record keeping from that period is incomplete and much disputed. Figures vary widely depending on which sources are used and how the historian is trying to measure them. In the case of Russia and Turkey, no system for tracking either military or civilian deaths existed, so it is impossible to determine them with any accuracy. Historian Modris Eksteins estimates that 9 million died, while historian John Keegan places the number at 10 million and Ian F.W. Becekett at 9 million to 10 million. Still others, like Fritz Haber’s son Ludwig Haber, use a percentage of the European population to estimate the dead. Modris Eksteins, Rites of Spring: The Great War and the Birth of the Modern Age (Boston: Houghton Mifflin, 1989); John Keegan, The First World War (New York: Alfred A. Knopf, 1999); Ian F.W. Beckett, The Great War, 1914–1918 (New York: Longman, 2001); Haber, The Poisonous Cloud, 31–39.

7. On the use of chemical weapons in the ancient world, see Mayor Adrienne, Greek Fire, Poison Arrows, and Scorpion Bombs: Biological and Chemical Warfare in the Ancient World (New York: Overlook Duckworth, 2003). On uses in the 19th century, see Edward M. Spiers, Chemical Warfare (Urbana: University of Illinois Press, 1986), 13–14. See also Joy, “Historical Aspects of Medical Defense,” 88–90.

8. The full text of the 1899 convention is available at http://www.yale.edu/lawweb/avalon/lawofwar/hague02.htm; the full text of the 1907 convention is available at http://www.yale.edu/lawweb/avalon/lawofwar/hague04.htm, both accessed January 15, 2007.

9. Recent interest in the life of Haber includes 2 new books: Dietrich Stozenberg, Fritz Haber: Chemist, Nobel Laureate, German, Jew (Philadelphia: Chemical Heritage Press, 2004), and Daniel Charles, Master Mind: The Rise and Fall of Fritz Haber, the Nobel Laureate Who Launched the Age of Chemical Warfare (New York: Ecco, 2005). For a brief intellectual biography of Fritz Haber, see W.A.E. McBryde, “1918 Nobel Laureate Fritz Haber,” in Nobel Laureates in Chemistry, 1901–1992, ed. Laylin K. James (Philadelphia: American Chemical Society and the Chemical Heritage Foundation, 1993), 114–123.

10. The concept of the “technological fix” is embedded in much of the history of technology. The idea that social, economic, and cultural problems can be solved quickly by applying technologies to them is most notably associated with the development of war technologies such as gas weapons, radar, and the gyroscope. However, as historians Hughes Thomas P. and Wiebe E. Bijker have illustrated, technological fixes are also an essential part of electrical systems and more everyday objects such as bicycles and light bulbs. On the technological fix, see Merritt Roe Smith and Leo Marx, Does Technology Drive History? The Dilemma of Technological Determinism (Cambridge, Mass: MIT Press, 1996), particularly the introduction. On the relationship between Hughes’s concepts of the technological fix and technological momentum, see Thomas P. Hughes, Networks of Power: Electrification in Western Society 1880–1930 (Baltimore: Johns Hopkins University Press, 1983), 140–174, 201–226. For technological fixes and large systems, see Hughes, Rescuing Prometheus (New York: Pantheon Books, 1998).

11. The mobilization of academic resources in Great Britain to solve the problem of gas warfare reflected the overall commitment of the country at large to prosecuting the war successfully. Scientists at Birmingham, Cambridge, Finsbury Technical College, Imperial College London, and St. Andrews carried out specific military research programs. Because British soldiers were initially unprepared for and unprotected from German gas attacks, defensive measures took first priority. The Army Medical College at Millbrook took the initial lead in developing protective devices, but soon physiology departments at Bedford College, the Lister Institute (including the animal station), Oxford University, the School of Agriculture in Cambridge, and University College London all contributed to the effort. See Haber, The Poisonous Cloud, 106–138.

12. For an expansive analysis of industrialization in World War I, see Beckett, The Great War. On the relationship between science and technology in World War I in the United States, the best work remains Daniel J. Kevles’s The Physicists: The History of a Scientific Community in Modern America (New York: Alfred A. Knopf, 1978); see especially 102–138 and 446–450. See also Daniel J. Kevles, “George Ellery Hale, the First World War, and the Advancement of Science in America,” Isis 59 (1968): 427–437. On the specific topic of research on chemical weapons during World War I, see Daniel P. Jones, “Chemical Warfare Research During World War I: A Model of Cooperative Research,” in Chemistry and Modern Society, ed. John Parascondola and James C. Whorton (Washington, DC: American Chemical Society, 1983). For a general introduction to the topic of technology and war in the United States, see Alex Roland, “Science and War,” Osiris, 1 (1985): 261–261; see also by Roland, “Technology and War: A Bibliographic Essay,” in Military Enterprise and Technological Change: Perspective on the American Experience, ed. Merritt Roe Smith (Cambridge, MA: MIT Press, 1985), 347–379. For a discussion of military innovation exclusive of chemical warfare in the period immediate following World War I, see Williamson Murray, “Innovation: Past and Future,” in Military Innovation in the Interwar Period, ed. Williamson Murray and Allan R. Millet (New York: Cambridge University Press, 1996), 300–328. On other aspects of chemical warfare research during the period, see Sarah Jansen, “Chemical-Warfare Techniques for Insect Control: Insect ‘Pests’ in Germany Before and After World War I,” Endeavour 24 (2000): 28–33.

13. Brophy and Fisher, The Chemical Warfare Service: Organizing for War, 3.

14. The National Research Council report is quoted in Brophy and Fisher, The Chemical Warfare Service, 4.

15. Ireland M.W., The Medical Department of the United States Army in the World War, Volume XIV: Medical Aspects of Gas Warfare (Washington, DC: Government Printing Office, 1926), 35–36. Ireland went on to list the organizations and facilities where war-related research was conducted; these included “the Bureau of Mines, Pittsburgh, PA; the National Carbon Co., Cleveland, OH; the Forest Products Laboratory, Madison, WI; the University of Chicago; the research laboratory of the American Sheet & Tin Plate Co., Pittsburgh, PA; the Bureau of Chemistry laboratory, Washington, DC; the Yale Medical School laboratory, New Haven, CT; the Massachusetts Institute of Technology, Cambridge, MA; the Mellon Institute, Pittsburgh, PA, and elsewhere. . . . Branch laboratories were organized from time to time at the Catholic University of American, Washington, DC; Johns Hopkins University, Baltimore, MD; Princeton University, Princeton, NJ; National Carbon Co., Cleveland, OH; Nela Park, Cleveland, OH; Harvard University, Cambridge, MA; Yale University, New Haven, CT; Wesleyan University, Middletown, CT; Ohio State University Columbus, OH; [Bryn Mawr College,] Bryn Mawr, PA; Massachusetts Institute of Technology, Cambridge, MA; Cornell University, Ithaca and New York City, NY; University of Michigan, Ann Arbor, MI; Clark University, Worcester, MA; Worcester Polytechnic Institute, Worcester, MA; University of Wisconsin, Madison, WI; Sprague Institute, Chicago, IL; and Ordnance Proving Ground, Lakehurst, NJ” (35–36). On the history of American research universities, see Roger L. Geiger, To Advance Knowledge: The Growth of American Research Universities in the Twentieth Century, 1900–1940 (New York: Oxford University Press, 1986). For an introduction to the history of chemists and specific scientific research schools in the United States, see John Servos, Physical Chemistry From Ostwald to Pauling: The Making of a Science in America (Princeton, NJ: Princeton University Press, 1990). See also John Servos, “Research Schools and Their Histories,” Osiris 8 (1993): 3–15; Robert E. Kohler, From Medical Chemistry to Biochemistry: The Making of a Biomedical Discipline (Cambridge, MA: Harvard University Press, 1982).

16. Ireland, Medical Department of the United States Army in the World War, 35.

17. James Conant became one of the architects of the academic–military–industrial complex in the United States during the postwar period. During World War II, Conant worked closely with Vannevar Bush in leading the Office of Scientific Research and Development. Both men were instrumental in directing much of the country’s military research during World War II, including the Manhattan Project. For a summary of his research activities during World War I, see Hershberg James G., James B. Conant: Harvard to Hiroshima and the Making of the Nuclear Age (New York: Alfred A. Knopf, 1993), 35–48. See also James B. Conant, My Several Lives: Memoirs of a Social Inventor (New York: Harper & Row, 1970), especially 41–53.

18. Slotten Hugh, “Humane Chemistry or Scientific Barbarism? American Responses to World War I Poison Gas, 1915–1930,” Journal of American History 77 (September 1990): 485. See also Ireland, Medical Department of the United States Army in the World War, 25. [Google Scholar]

19. Haber, The Poisonous Cloud, 107–138.

20. Jon Silkin, The War Poems: Wilfred Owen (London: Sinclair-Stevenson, 1994), 24. Owen was killed in action on November 4, 1918 at the Sambre-Oise Canal, a week before the armistice, by bullet, not by gas. For an introduction to the cultural and literary ramifications of Owen’s war poetry see: Tim Kendall, Modern English War Poetry, (New York : Oxford University Press, 2006); Daniel W. Hipp, The Poetry of Shell Shock: Wartime Trauma and Healing in Wilfred Owen, Ivor Gurney and Siegfried Sassoon (Jefferson, NC: McFarland & Co., 2005); and Santanu Das, Touch and intimacy in First World War Literature, (New York: Cambridge University Press, 2005).

21. On the life of Walther Nerst, see McBryde W.A.E., “Walther Herman Nernst,” in Nobel Laureates in Chemistry, 1901–1992, ed. Laylin K. James (Philadelphia: American Chemical Society and the Chemical Heritage Foundation, 1993), 125–133.

22. Moore William, Gas Attack! Chemical Warfare 1915–1918 and Afterwards (New York: Hippocrene Books, 1987), 12.

23. Haber, The Poisonous Cloud, 51, 177.

24. Watkins, Methodist Report.

25. Palazzo Albert, Seeking Victory on the Western Front: The British Army and Chemical Warfare in World War I (Lincoln: University of Nebraska Press, 2000), 42.

26. Arthur Max, Last Post (London: Weidenfeld & Nicolson, 2005), 35–36. See also Palazzo, Seeking Victory on the Western Front, 42–43.

27. On gas attacks, see Goss B.C., “An Artillery Gas Attack,” Journal of Industrial and Engineering Chemistry 11 (September 1919): 829–836; James C. Webster, “The First Gas Regiment,” Journal of Industrial and Engineering Chemistry 11 (July 1919): 621–629; Army War College, Gas Warfare, Part 2: Methods of Defense Against Gas Attacks (Washington, DC: War Department, 1918). For interesting views on gas warfare from earlier periods, see Edward S. Farrow, Gas Warfare (New York: E.P. Dutton & Company, 1920); War Office, Medical Manual of Chemical Warfare (London: His Majesty’s Stationary Office, 1939); Alden H. Waitt, Gas Warfare: The Chemical Weapon, Its Use, and Protection Against It (New York: Duell, Sloan and Pearce, 1942). On the design of gas masks and respirators, see P.W. Carleton, “Anti-Dimming Compositions for Use in the Gas Mask,” Journal of Industrial and Engineering Chemistry 11 (December 1919): 1105–1111; J. Perrot, Max Yablick, and A.C. Fieldner, “New Absorbent for Ammonia Respirators,” Journal of Industrial and Engineering Chemistry 11 (November 1919): 1013–1019; Army War College, Methods of Defense Against Gas Attacks. See also in that series: Army War College, Gas Warfare, Part 1: German Methods of Offense (Washington, DC: War Department, 191 8); Army War College, Gas Warfare, Part 3: Methods of Training in Defensive Measures (Washington, DC: War Department, 1918); Army War College, Gas Warfare, Part 4: The Offensive in Gas Warfare, Cloud and Projector Attacks (Washington, DC: War Department, 1918); Palazzo, Seeking Victory on the Western Front, 42–43. [Google Scholar]

28. Palazzo, Seeking Victory on the Western Front, 43. See also Haber, The Poisonous Cloud, 46–46.

29. Haber, The Poisonous Cloud, 47.

30. Winter Denis, Death’s Men: Soldiers in the Great War (New York: Penguin Books, 1979), 124; also cited in Joy, “Historical Aspects of Medical Defense,” 92.

31. Arthur, Last Post, 35–36.

32. Joy, “Historical Aspects of Medical Defense,” 97. For information regarding current military procedures for dealing with chemical warfare causalities and chemical warfare defense, see the following in Medical Aspects of Chemical and Biological Warfare: Frederick R. Sidell, Ronald R. Bresell, Robert H. Mosebar K Mills McNeill, and Ernest T. Takafuji, “Field Management of Chemical Casualties” (325–336); Frederick R. Sidell, “Triage of Chemical Casualties” (337–350); Charles G. Hurst, “Decontamination” (351–360); Michael R. O’Hern, Thomas R. Dashiell, and Mary Frances Tracy, “Chemical Defense Equipment” (361–397).

33. Haber suggests that the actual figures were significantly higher; see The Poisonous Cloud, 248.

34. Joy, “Historical Aspects of Medical Defense,” 91.

35. The report is quoted in Ireland, Medical Department of the United States Army in the World War, 27.

36. Ibid, 28.

37. Ibid, 28, 33–37.

38. Eksteins, Rites of Spring, 162.

39. Winter, Death’s Men, 121.

40. Quoted in Cochrane Rexmond C., Third Division at Thierry July, 1918 (Army Chemical Center, MD: US Army Chemical Corps Historical Office, 1959), 91.

41. Joy, “Historical Aspects of Medical Defense,” 94.

42. A 1919 article by Gross B.C. of the Chemical Warfare Service recommended attacking in wind conditions of less than 3 miles per hour at a relative humidity of 40% to 50%. The author noted that having considered “temperature, wind, and humidity conditions, the hours between midnight and daylight are usually the most favorable for a gas attack, and, in addition, surprise is more easily possible at this time.” Successful gas-based artillery barrages were designed to hit “small, definitely located targets known to be occupied [with a] concentrated fire of 2 minutes duration.” B.C. Goss, “An Artillery Gas Attack,” The Journal of Industrial and Engineering Chemistry 11 (September 1919): 831. [Google Scholar]

43. Joy, “Historical Aspects of Medical Defense,” 100.

44. Gilchrist Harry L. and Philip B. Matz, The Residual Effects of Wartime Gases (Washington, DC: Government Printing Office; 1933), 44; also cited in Joy, “Historical Aspects of Medical Defense,” 96.

45. German attacks employing mustard gas were extremely difficult for all Allied troops, especially newly arriving AEF. Unaccustomed to gas attacks under combat conditions with even chlorine and phosgene, the AEF moved to the front just as gas warfare entered its most violent and destructive phase. In addition, an earlier decision by American scientific, medical, and military leaders to focus almost exclusively on the gas mask in lieu of a more complete approach to gas defense and treatment may have been ill conceived. A 1926 US Army medical history of gas warfare noted that the “Medical Department of the United States Army with respect to gas warfare were [sic] concerned with furnishing gas masks and other prophylactic apparatus for the Army, rather than with preparation for the care and treatment of gas casualties.” Ireland, Medical Department of the United States Army in the World War, 27.

46. August M. Prentiss, Chemicals in Warfare (New York: McGraw-Hill Book Company, 1937), 579. On the environmental consequences of war, see also Richard P. Tucker and Edmund Russell Natural Enemy, Natural Ally: Toward an Environmental History of War, ed. (Corvallis, OR: Oregon State University Press, 2004).

47. Joy, “Historical Aspects of Medical Defense,” 98. On the medical aspects of vesicants in general and mustard gas in particular, see Frederick R Sidell, John S. Urbanetti, William J. Smith, and Charles G. Hurst, “Vesicants,” in Medical Aspects of Chemical and Biological Warfare, 197–228; Frederick R. Sidell and Charles G. Hurst, “Long-Term Heal Effects of Nerve Agents and Mustard,” in Medical Aspects of Chemical and Biological Warfare, 229–246.

48. Joy, “Historical Aspects of Medical Defense,” 101.

49. Arthur, Last Post, 83.

50. Joy, “Historical Aspects of Medical Defense,” 98.

51. For a full history of the development of lewisite, see Vilensky Joel A., Dew of Death: The Story of Lewisite, America’s World War I Weapon of Mass Destruction (Bloomington: Indiana University Press, 2005).

52. These figures are based on Gilchrist Harry L., A Comparative Study of World War Casualties From Gas and Other Weapons (Edgewood Arsenal, MD: Chemical Warfare School, 1928), p. 21, Table 7.

53. Spiers, Chemical Warfare, 13. On the general history of chemical manufacturing and chemical engineering in the United States, see John Kenly Smith, Jr, “The Evolution of the Chemical Industry: A Technological Perspective,” in Chemical Sciences in the Modern World, ed. Seymour H. Mauskopf (Philadelphia: University of Pennsylvania Press, 1993). For a view of chemical engineering activity during World War I, see Charles O. Brown, “US Chemical Plant for Manufacturing Sodium Cyanide, Saltville, Virginia,” Journal of Industrial and Engineering Chemistry 11 (November 1919): 1010–1013; James F. Norris, “The Manufacture of War Gases in Germany,” Journal of Industrial and Engineering Chemistry 11 (September 1919): 817–829; Edward H. Hempel, The Economics of Chemical Industries (New York: John Wiley & Sons, 1939), 28–31.

54. Ryan T. Anthony, Christien Ryan, Elaine A. Seddon, and Kenneth R. Seddon, Phosgene and Related Carbonyl Halides (New York: Elsever, 1996), 12–13.

55. Ibid, 15–16.

56. Norris, “Manufacture of War Gases in Germany,” 823–824. Norris was one of a group of Allied officers who toured the occupied zones along the Rhine after the war to gather intelligence (both military and industrial) on the state of the German chemical industry. His report on the Bayer Company plant at Höchst, which manufactured per-chloromethylformate (or superpalite), is intriguing, as it offers a window into one of the best designed and “safest” chemical production facilities in Germany. Norris writes that “In the early days they had several small explosions which were not serious. They had a large number of cases of poisoning among the workmen, which were also not serious. All the workers were supplied with gas masks and put them on in case of emergency only. The workers suffered from chest and heart affections. Two or three died during the time [sic] though our informant would not admit this was necessarily the cause of death. They sometimes had as many as one-third of the workers absent owing to sickness. The workers were not changed as they wished to retain those who could stand it. Only men were employed in the plant.” Norris, 824.

57. Ibid, 824. See also Haber, The Poisonous Cloud, 257–258. The German military relied on the methods of Victor Meyer involving the preparation of thioglycol by the Badische Company, which was later converted to mustard gas at the Bayer Company plant at Höchst. Norris, 821–822.

58. Haber, The Poisonous Cloud, 251.

59. Ibid.

60. Ibid, 252.

61. Norris, “Manufacture of War Gases in Germany,” 824. See also Haber, The Poisonous Cloud, 257–258.

62. Haber, The Poisonous Cloud, 250–251.

63. “At first one man reported sick for every nine shells filled! . . . [B]etween 21 June and 7 December 1918, 1,213 casualties were recorded, the weekly rate occasionally reaching 100 per cent of the labour force. . . . Avonmouth and Chittening between them (there are no figures for Banbury) reported altogether 2,600 gas cases.” Haber, The Poisonous Cloud, 251. See also Industrial Fatigue Research Board, The Output of Women Workers in Relation to Hours of Work in Shell-Making, Report No. 2 (London: His Majesty’s Stationary Office, 1919).

64. Ireland, Medical Department of the United States Army in the World War, 32–33. The surgeon general’s responsibilities involved far more than local plant supervision. They included a research service responsible for work on the “chronic effects of low-concentration war gases, protective devices and therapy”; a field service responsible for work on the “selection, appointment, and training of contract surgeons and medical officers, installation of emergency ward[s], dispensary and first-aid equipment, and inspection”; and an education service that “collect[ed] reports, case histories, and information, prepar[ed] bulletins of information for medical officers and contracts surgeons, assign[ed] problems for solutions in laboratories, and develop[ed] special course[s] of instruction at American University for medical officers, to be used later in factory instruction and assigned to the large plants for duty.” Ibid, 33.

65. Ibid, 25.

66. Haber, The Poisonous Cloud, 252.

67. Arthur, Last Post, 35–36.

68. The text of the Geneva Protocol is available from the US Department of State at http://www.state.gov/t/ac/trt/4784.htm#treaty, accessed January 17, 2007. For a detailed examination of these international conferences, see Spiers, Chemical Warfare, 41–47; Victor A Utgoff, The Challenge of Chemical Weapons: An American Perspective (New York: Macmillan, 1990), 14–21.

69. For an introduction to the US strategic bombing campaigns during World War II and the public health ramifications of large-scale urban destruction, see Sherry Micheal S., The Rise of American Air Power: The Creation of Armageddon (New Haven, CT: Yale University Press, 1987); Tami Davis Biddle, “British and American Approaches to Strategic Bombing: Their Origins and Implementation in the World War II Combined Bomber Offensive,” in Airpower: Theory and Practice, ed. John Gooch (London, England: Frank Cass & Co, 1995), 91–144; See also by Biddle, Rhetoric and Reality in Air Warfare (Princeton, NJ: Princeton University Press, 2002); Hermann Knell, To Destroy a City: Strategic Bombing and Its Human Consequences in World War II (Cambridge, MA: Da Capo Press, 2003); Alvin D. Coox, “Strategic Bombing in the Pacific, 1942–1945,” in Case Studies in Strategic Bombardment, ed. R. Cargill Hall (Washington, DC: Air Force History and Museums Program, 1998), 253–382; Kenneth Hewitt, “Place Annihilation: Area Bombing and the Fate of Urban Places,” Annals of the Association of American Geographers 73 (1983): 257–284. Finally, it is important to keep in mind that the US military’s invasion plans for Japan (Operation Downfall), originally scheduled to commence in November 1945, included provisions for the tactical and strategic use of atomic, biological, and chemical weapons against various military and civilian targets.

70. Throughout the 20th century, the number of civilian deaths increased at a startling rate. Only 14% of deaths during World War I were civilians; by contrast, the rate in World War II was 67%; by the 1980s the rate was 75%, and in the 1990s it reached 90%. Garfield and Neugut, “The Human Consequences of War,” Table 3–3, 31.

71. On the nature of biological weapons programs during World War II, see Fitzgerald Gerard J., “Babies, Barriers, and Bacteriological Engineers: Biological Warfare Research at LOBUND, 1930–1952,” Technology and Culture, in press; Edward M. Eitzen, “Historical Overview of Biological Warfare,” in Medical Aspects of Chemical and Biological Warfare, 415–423. For an introduction to biological and chemical warfare research activities after World War II, see Gradon Carter and Brian Balmer, “Chemical and Biological Warfare and Defence, 1945–1990,” in Cold War, Hot Science: Applied Research in Britain’s Defence Laboratories, 1945–1990, ed. Robert Bud and Philip Gummett (Amsterdam, The Netherlands: Harwood Academic Publishers, 1999), 295–338. See also G.B. Carter and G.S. Pearson, “North Atlantic Chemical and Biological Research Collaboration: 1916–1995,” Journal of Strategic Studies 19 (March 1996): 74–103; Mark Wheelis, Lajos Rózsa, and Malcolm Dando, Deadly Cultures: Biological Weapons Since 1945 (Cambridge, MA: Harvard University Press, 2006).

72. For a general overview of chemical weapons development in the United States, see Smart, “History of Chemical and Biological Warfare: An American Perspective,” in Medical Aspects of Chemical and Biological Warfare. On the postwar development of nerve agents, see Frederick R. Sidell, “Nerve Agents,” in Medical Aspects of Chemical and Biological Warfare, 129–179; Michael A. Dunn, Brennie E. Hackley Jr, and Fred-erick R. Sidell, “Pretreatment for Nerve Agent Exposure,” in Medical Aspects of Chemical and Biological Warfare, 181–196; Jonathan B. Tucker, War of Nerves: Chemical Warfare From World War I to Al-Qaeda (New York: Pantheon, 2006), 158. For an analysis of nerve agent development during the cold war, see Bradley S. Davis, “Transitional Perspectives on Conventional, Chemical and Biological Weapons Production,” in United States Post–Cold War Defense Interests: A Review of the First Decade, ed. Karl P. Magyar (New York: Palgrave Macmillan, 2004), 136. For an examination of contemporary debates over chemical weapons destruction in relation to public health, see Michael Greenberg, “Public Health, Law, and Local Control: Destruction of the US Chemical Weapons Stockpile,” American Journal of Public Health 93 (2003): 1222–1226 and Smart, “History of Chemical and Biological Warfare: An American Perspective.”

73. For fascinating views on the future of gas warfare as seen by contemporary observers during the interwar period, see Farrow Edward S., Gas Warfare (New York: E.P. Dutton & Company, 1920), 166; War College, “Aspects in Which the Use of Gas Is Likely to Differ in a Future War,” in Medical Manual of Chemical Warfare (London: His Majesty’s Stationary Office, 1939).

74. Conant James B., My Several Lives: Memoirs of a Social Inventor (New York: Harper & Row, 1970), 49.

75. Hermann Armin, The New Physics: The Route Into the Atomic Age (Munich: Heinz Moos Verlag, 1979), 47, 73.


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Chemical Warfare Agents Developed During World War I

Chlorine (Cl2)Phosgene: Carbonyl Chloride (COCl2)Mustard Gas: ββ’-Dichlorethyl Sulfide (ClCH2CH2)2SLewisite: β-Chlorovinyldichloroarsine (ClCH=CHAsCl2)
US Army chemical warfare service symbolClCGHSM-1
Physiological classificationLung injurantLung injurantVesicantVesicant
Tactical classificationCasualty agentCasualty agentCasualty agentCasualty agent
Vapor density compared with air2.53.55.57.1
PersistencySummer: 5 min in open, 20 min in woods; winter: 10 min in open, 60 min in woodsSummer: 10 min in open 3 min in woods; winter: 20 min in open, 2 h in woodsSummer: 24 h in open, 1 wk in woods; winter: several weeks in both the open and woodsSummer: 24 h in open, 1 wk in woods; winter: 1 wk in the open and woods
Lethal concentration, mg/L or oz/1000 cu ft/10 min exposure30-min exposure: 2.53; 10-min exposure: 5.6030-min exposure: 0.36; 10-min exposure: 0.5030-min exposure: 0.07; 10-min exposure: 0.1530-min exposure: 0.48; 10-min exposure: 0.12
OdorPungentFresh-cut hayGarlic or horseradishLike geraniums, then biting
NeutralizationAlkali solution or solidSteam will hydrolyze; alkalis and amines react with CGBleaching powder 3% solution sodium sulfide (Na2S) in water; steam; gaseous chlorine; or bury under moist earthAlcoholic sodium hydroxide spray
Physiological actionBurns upper respiratory tractBurns lower lung surfaces causing edemaDissolves in skin and then produces burnsDissolves in skin then burns and liberates M-1 oxide, which poisons body
ProtectionGas masks, absorbents in canisters onlyGas masks, absorbents in canisters onlyGas masks and protective clothingGas masks and best of protective clothing
First aidKeep patient quiet and warm and treat for bronchial pneumoniaKeep patient calm; administer heart stimulants; give oxygen in severe cases, treat like pleurisyWash affected parts with kerosene or gasoline, then with strong soap and water; rub, dry, rinse with hot clean water; agent must be removed within 3 min Eye casualty concentration 1 h exposure, .0001 (mg liter).Wash with oils, hot water, and soap; dry; first aid must be applied at once